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Home > Doctors > Ethan J. Weiss

Ethan J. Weiss, M.D.

Cardiologist

Dr. Ethan Weiss is a cardiologist specializing in general cardiology. His special interests include preventive cardiology, genetics of coronary disease, risk assessment and heart disease in the young. In his research, Weiss uses genetic models to better understand the mechanism of metabolic disorders such as obesity, fatty liver disease, and diabetes. He also studies the blood clotting system and has interest in identifying novel ways to safely block clots associated with diseases such as heart attack and stroke without causing an increase in bleeding.

Weiss earned a medical degree at Johns Hopkins University School of Medicine. He completed an internship and residency on the Osler Medical Service of the Johns Hopkins Hospital. He came to the University of California, San Francisco in 1998 as a cardiology fellow and is currently an assistant professor of medicine.

Read a Q&A with Weiss.

Clinics

Cardiovascular Care and Prevention Center at Mission Bay
535 Mission Bay Blvd. South
San Francisco, CA 94158
Phone: (415) 353-2873
Fax: (415) 353-2528

Hours: Monday to Friday
8 a.m. – 5 p.m.

Center for Prevention of Heart and Vascular Disease
535 Mission Bay Blvd. South
San Francisco, CA 94158
Main Phone: (415) 353-2873
First Appointment: (415) 514-6621
Follow-up Visit: (415) 514-6620
Fax: (415) 353-2528

Hours: Monday to Friday
8 a.m. – 5 p.m.

Conditions & Treatments

More about Ethan J. Weiss

Education

Johns Hopkins University School of Medicine 1996

Residencies

Johns Hopkins Hospital, Internal Medicine 1998

Fellowships

UCSF Medical Center, Cardiology 2003

Selected Research and Publications

  1. Hsue PY, Scherzer R, Grunfeld C, Nordstrom SM, Schnell A, Kohl LP, Nitta E, Martin JN, Deeks SG, Weiss EJ. HIV infection is associated with decreased thrombin generation. Clin Infect Dis. 2012 Apr; 54(8):1196-203.
  2. Nordstrom SM, Tran JL, Sos BC, Wagner KU, Weiss EJ. Liver-derived IGF-I contributes to GH-dependent increases in lean mass and bone mineral density in mice with comparable levels of circulating GH. Mol Endocrinol. 2011 Jul; 25(7):1223-30.
  3. Sos BC, Harris C, Nordstrom SM, Tran JL, Balázs M, Caplazi P, Febbraio M, Applegate MA, Wagner KU, Weiss EJ. Abrogation of growth hormone secretion rescues fatty liver in mice with hepatocyte-specific deletion of JAK2. J Clin Invest. 2011 Apr; 121(4):1412-23.
  4. Williams MS, Weiss EJ, Sabatine MS, Simon DI, Bahou WF, Becker LC, Parise LV, Dauerman HL, French PA, Smyth SS, Becker RC. Genetic regulation of platelet receptor expression and function: application in clinical practice and drug development. Arterioscler Thromb Vasc Biol. 2010 Dec; 30(12):2372-84.
  5. Nordstrom SM, Holliday BA, Sos BC, Smyth JW, Levy RE, Dukes JW, Lord ST, Weiss EJ. Increased thrombosis susceptibility and altered fibrin formation in STAT5-deficient mice. Blood. 2010 Dec 16; 116(25):5724-33.
  6. Nordstrom SM, Weiss EJ. Sex differences in thrombosis. Expert Rev Hematol. 2008 Oct; 1(1):3-8.
  7. Wong JH, Dukes J, Levy RE, Sos B, Mason SE, Fong TS, Weiss EJ. Sex differences in thrombosis in mice are mediated by sex-specific growth hormone secretion patterns. J Clin Invest. 2008 Aug; 118(8):2969-78.
  8. Weiss EJ, Hamilton JR, Lease KE, Coughlin SR. Protection against thrombosis in mice lacking PAR3. Blood. 2002 Nov 1; 100(9):3240-4.
  9. Sambrano GR, Weiss EJ, Zheng YW, Huang W, Coughlin SR. Role of thrombin signalling in platelets in haemostasis and thrombosis. Nature. 2001 Sep 6; 413(6851):74-8.
  10. Shapiro MJ, Weiss EJ, Faruqi TR, Coughlin SR. Protease-activated receptors 1 and 4 are shut off with distinct kinetics after activation by thrombin. J Biol Chem. 2000 Aug 18; 275(33):25216-21.
  11. Faruqi TR, Weiss EJ, Shapiro MJ, Huang W, Coughlin SR. Structure-function analysis of protease-activated receptor 4 tethered ligand peptides. Determinants of specificity and utility in assays of receptor function. J Biol Chem. 2000 Jun 30; 275(26):19728-34.
  12. Nakanishi-Matsui M, Zheng YW, Sulciner DJ, Weiss EJ, Ludeman MJ, Coughlin SR. PAR3 is a cofactor for PAR4 activation by thrombin. Nature. 2000 Apr 6; 404(6778):609-13.
  13. Goldschmidt-Clermont PJ, Coleman LD, Pham YM, Cooke GE, Shear WS, Weiss EJ, Kral BG, Moy TF, Yook RM, Blumenthal RS, Becker DM, Becker LC, Bray PF. Higher prevalence of GPIIIa PlA2 polymorphism in siblings of patients with premature coronary heart disease. Arch Pathol Lab Med. 1999 Dec; 123(12):1223-9.
  14. Bray PF, Weiss EJ, Tayback M, Goldschmidt-Clermont PJ. PlA1/A2 polymorphism of platelet glycoprotein IIIa and risk of cardiovascular disease. Lancet. 1997 Apr 12; 349(9058):1100-1.
  15. Vandervoort J, Weiss EJ, Somnay K, Tham TC, Wong RC, Carr-Locke DL. Self-expanding metal stent for obstructing adenocarcinoma of the sigmoid. Gastrointest Endosc. 1996 Dec; 44(6):739-41.
  16. Weiss EJ, Bray PF, Tayback M, Schulman SP, Kickler TS, Becker LC, Weiss JL, Gerstenblith G, Goldschmidt-Clermont PJ. A polymorphism of a platelet glycoprotein receptor as an inherited risk factor for coronary thrombosis. N Engl J Med. 1996 Apr 25; 334(17):1090-4.
  17. Weiss EJ, Goldschmidt-Clermont PJ, Grigoryev D, Jin Y, Kickler TS, Bray PF. A monoclonal antibody (SZ21) specific for platelet GPIIIa distinguishes P1A1 from P1A2. Tissue Antigens. 1995 Nov; 46(5):374-81.

Publications are derived from MEDLINE/PubMed and provided by UCSF Profiles, a service of the Clinical & Translational Science Institute (CTSI) at UCSF. Researchers can make corrections and additions by logging on to UCSF Profiles.

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