"Fibrosis is a big problem. If you improve people and slow the progression of their disease, that’s a success."
Where I see patients (2)
My reviews
Selected research
-
An in vivo screening platform identifies senolytic compounds that target p16INK4a+ fibroblasts in lung fibrosis.
The Journal of clinical investigation
-
Genome-wide screens identify SEL1L as an intracellular rheostat controlling collagen turnover.
Nature communications
-
Reply: Telomere length and immunosuppression in non-idiopathic pulmonary fibrosis interstitial lung disease.
The European respiratory journal
5.0