James Maas, PhD, MD


Dr. James Maas is a neurologist who specializes in treating patients with movement disorders, including Parkinson's disease, essential tremor and dystonia. He sees patients at the Movement Disorder and Neuromodulation Center at Mount Zion.

Maas' research centers on the role of proteins implicated in Parkinson's disease, as well as molecules that determine function in dopamine neurons.

Maas received his medical degree from the Washington University School of Medicine in St. Louis, where he also completed a doctorate in neuroscience. He then completed a residency in neuroscience at UCSF.

A clinical professor of neurology, Maas belongs to the American Academy of Neurology.


Movement Disorder and Neuromodulation Center
1635 Divisadero St., Suite 520
San Francisco, CA 94115
Neurology: (415) 353-2311
Neurosurgery: (415) 353-2071
Fax: (415) 353-9060

Hours: Monday to Friday
8 a.m. – 5 p.m.

Board Certification

Neurology, American Board of Psychiatry and Neurology

Academic Title

Assistant Professor

More about James Maas


Washington State University, M.D.; Doctorate of Philosophy, Neuroscience 2006


UCSF Medical Center 2010


UCSF, Neurology 2012

Selected Research and Publications

  1. Maas JW, Yang J, Edwards RH. Endogenous Leucine-Rich Repeat Kinase 2 Slows Synaptic Vesicle Recycling in Striatal Neurons. Front Synaptic Neurosci. 2017; 9:5.
  2. Asensio CS, Sirkis DW, Maas JW, Egami K, To TL, Brodsky FM, Shu X, Cheng Y, Edwards RH. Self-assembly of VPS41 promotes sorting required for biogenesis of the regulated secretory pathway. Dev Cell. 2013 Nov 25; 27(4):425-37.
  3. Maas JW. Inherited myelopathies. Semin Neurol. 2012 Apr; 32(2):114-22.
  4. Lee HY, Huang Y, Bruneau N, Roll P, Roberson ED, Hermann M, Quinn E, Maas J, Edwards R, Ashizawa T, Baykan B, Bhatia K, Bressman S, Bruno MK, Brunt ER, Caraballo R, Echenne B, Fejerman N, Frucht S, Gurnett CA, Hirsch E, Houlden H, Jankovic J, Lee WL, Lynch DR, Mohammed S, Müller U, Nespeca MP, Renner D, Rochette J, Rudolf G, Saiki S, Soong BW, Swoboda KJ, Tucker S, Wood N, Hanna M, Bowcock AM, Szepetowski P, Fu YH, Ptácek LJ. Mutations in the gene PRRT2 cause paroxysmal kinesigenic dyskinesia with infantile convulsions. Cell Rep. 2012 Jan 26; 1(1):2-12.
  5. Wieczorek L, Maas JW, Muglia LM, Vogt SK, Muglia LJ. Temporal and regional regulation of gene expression by calcium-stimulated adenylyl cyclase activity during fear memory. PLoS One. 2010 Oct 14; 5(10):e13385.
  6. Conti AC, Maas JW, Moulder KL, Jiang X, Dave BA, Mennerick S, Muglia LJ. Adenylyl cyclases 1 and 8 initiate a presynaptic homeostatic response to ethanol treatment. PLoS One. 2009 May 27; 4(5):e5697.
  7. Conti AC, Maas JW, Muglia LM, Dave BA, Vogt SK, Tran TT, Rayhel EJ, Muglia LJ. Distinct regional and subcellular localization of adenylyl cyclases type 1 and 8 in mouse brain. Neuroscience. 2007 May 11; 146(2):713-29.
  8. Maas JW, Vogt SK, Chan GC, Pineda VV, Storm DR, Muglia LJ. Calcium-stimulated adenylyl cyclases are critical modulators of neuronal ethanol sensitivity. J Neurosci. 2005 Apr 20; 25(16):4118-26.
  9. Maas JW, Indacochea RA, Muglia LM, Tran TT, Vogt SK, West T, Benz A, Shute AA, Holtzman DM, Mennerick S, Olney JW, Muglia LJ. Calcium-stimulated adenylyl cyclases modulate ethanol-induced neurodegeneration in the neonatal brain. J Neurosci. 2005 Mar 02; 25(9):2376-85.
  10. Wei F, Qiu CS, Kim SJ, Muglia L, Maas JW, Pineda VV, Xu HM, Chen ZF, Storm DR, Muglia LJ, Zhuo M. Genetic elimination of behavioral sensitization in mice lacking calmodulin-stimulated adenylyl cyclases. Neuron. 2002 Nov 14; 36(4):713-26.
  11. Huang YH, Maas JW. D-Amphetamine at low doses suppresses noradrenergic functions. Eur J Pharmacol. 1981 Nov 05; 75(4):187-95.
  12. Huang YH, Maas JW, Hu GH. The time course of noradrenergic pre- and postsynaptic activity during chronic desipramine treatment. Eur J Pharmacol. 1980 Nov 07; 68(1):41-7.
  13. Maas JW, Huang Y. Noradrenergic function and depression, too much or too little? Can J Neurol Sci. 1980 Aug; 7(3):267-8.
  14. Redmond DE, Huang YH, Snyder DR, Maas JW, Baulu J. Hyperphagia and hyperdipsia after locus coeruleus lesions in the stumptailed monkey. Life Sci. 1977 May 01; 20(9):1619-28.
  15. Redmond DE, Huang YH, Snyder DR, Maas JW. Behavioral effects of stimulation of the nucleus locus coeruleus in the stump-tailed monkey Macaca arctoides. Brain Res. 1976 Nov 12; 116(3):502-10.
  16. Huang YH, Maas JW. Stimulation of the dorsal noradrenergic dundle and field potentials in the locus coeruleus. Brain Res. 1976 Oct 08; 115(1):91-7.
  17. Huang YH, Redmond DE, Snyder DR, Maas JW. In vivo location and destruction of the locus coeruleus in the stumptail macaque (Macaca arctoides). Brain Res. 1975 Dec 12; 100(1):157-62.

Publications are derived from MEDLINE/PubMed and provided by UCSF Profiles, a service of the Clinical & Translational Science Institute (CTSI) at UCSF. Researchers can make corrections and additions to their publications by logging on to UCSF Profiles.